Rosuvastatin is metabolized to a limited extent by CYP2C9 to form an N-desmethyl metabolite and the other metabolite is the rosuvastatin lactone.

The acid moeity (highlighted part) is likely to undergo glucuronidation first and subsequently lactonisation to form the rosuvastatin lactone
Unlike other statins, rosuvastatin undergoes relatively little metabolism by hepatic CYPs with no significant involvement of CYP3A4.
The metabolism of statins is complex, involving the inter-conversion
of acid/lactone by various pathways. The acid forms are converted to
the corresponding lactones via a glucuronide intermediate.
Here is the proposed, generalised metabolic pathway of statins. The acid forms of statins are converted to the corresponding lactones via a glucuronide intermediate via an elimination reaction.

Sidenote for some statins: (After metabolism by CYP3A4 some of the resulting lactone metabolites are converted to the corresponding acid forms:
References